Which of the following actions is NOT associated with Angiotensin II?

Angiotensin II is a key regulatory peptide in the renin-angiotensin-aldosterone system (RAAS), primarily known for its role in maintaining blood pressure and fluid balance. One of its main actions is to stimulate sodium reabsorption in the kidneys, which helps to increase blood volume and pressure. Additionally, Angiotensin II triggers the release of aldosterone from the adrenal cortex, further facilitating sodium retention and increasing potassium excretion.

The action of causing vasodilation of efferent arterioles is contrary to the physiological role of Angiotensin II. Instead, this peptide typically causes constriction of both afferent and efferent arterioles in the renal system, which can help to increase glomerular filtration pressure despite its overall vasoconstrictive effects, serving different functions in various vascular beds. By constricting the efferent arterioles, Angiotensin II helps maintain glomerular filtration rate in the face of low blood pressure.

Therefore, the choice indicating that Angiotensin II causes vasodilation of efferent arterioles is not aligned with its known biological effects.